[39:45] Actual infections
We classify 83% of cases as "mild" because they don't require hospitalization
What most likely happens is the virus destroys epithelium, exposing alveoli to fluid, and the balloons that transmit oxygen to your bloodstream become water balloons
Repair system can overreact and create too many layers between alveoli and capillaries, oxygen can't diffuse, and we're back to suffocation
This is the same phenotype as SARS-1 and MERS, as well as others
NIH has spend on the order of $108 trying to figure out how to treat this sort of end-stage lung disease; mortality rates have gone from 50% to 25–30%.
We aren't seeing secondary bacterial infections
Cytokine storms play a part in this process
We don't know why young children don't show symptoms; they certainly have the virus at high concentration
We don't know whether children are superspreaders; we're currently researching that
General coronaviruses infect young children and cause mild disease; maybe that immunity saves us as adults?
If true, this means we're facing a 1–3-year pandemic until herd immunity occurs
We don't see cross-immunity from similar coronaviruses
There are other coronaviruses in animal reservoirs that are posed to jump to humans.
It's just a matter of time until a human comes into contact with such an animal, by e.g. collecting guano
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